Scientist are still uncertain as to the actual cause of diabetes.
There are risk factors that make a person have a propensity to being diagnosed with the disease more than the average person.
As far as factors that are suspect to that control is weight.
If you are obese, the single best thing you can do for your health and the prevention of diabetes is to lose weight.
Even in small increments when we lose weight we improve our prognosis and outcome in regards to the disease.
These may be easier said than done.
There are new studies that are now showing that there is a genetic factor or mutation for people who are obese and have diabetes.
This genetic malfunction affects how the bodies use energy and insulin - two key elements in the functioning of your body and the cause of diabetes and obesity.
Genetic factors play an important role in type 2 diabetes, but the pattern is complicated, since both impairment of beta cell function and an abnormal response to insulin are involved.
Researchers have identified a number of genetic suspects: Researchers have identified genes responsible for maturity-onset diabetes in youth, a rare genetic form of type 2 diabetes that develops only in Caucasian teenagers.
Some research is now investigating genes that may be responsible for inherited cases of type 2 diabetes in middle-aged Caucasians.
A defective fatty-acid binding protein 2 gene may result in higher levels of unhealthy fat molecules which may be critical in the link between obesity and insulin resistance in some people with diabetes type 2.
A defective lipoprotein lipase gene may pose a risk for coronary artery disease and type 2 diabetes in people who have it.
Variations in a gene that regulates a protein called calpain-10 is proving to affect insulin secretion and action and may play a role in diabetes type 2.
There is some disagreement, however, about its significance.
Defective genes that regulate a molecule called peroxisome proliferator-activated receptor gamma may contribute to both type 2 diabetes and high blood pressure in some patients.
A defective gene has been detected that reduces activity of a protective substance called beta3-adrenergic receptor, which is found in visceral fat cells.
The result is a slow-down in metabolism and an increase in obesity.
The defective gene has been found in Pima Indians and other populations with a very high incidence of type 2 diabetes and obesity.
Much of the data is still in its early stages so time is still needed to evaluate the research.
For now, preventative measures and diet and weight control are the primary natural ways to avoid manifestation of the disease.
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